Polyarteritis nodosa risk factors
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Sargun Singh Walia M.B.B.S.[2]
Overview
Common risk factors in the development of PAN include hepatitis B virus infection and age 40 to 60. Less common risk factors in the development of PAN include hairy cell leukemia, hepatitis C virus and male sex.
Risk Factors
Common Risk Factors
- Common risk factors in the development of PAN include:
- Hepatitis B virus (HBV) infection[1][2]
- Age 40 to 60 years.
Less Common Risk Factors
- Less common risk factors in the development of PAN include:
- Hairy cell leukemia[3]
- Blood transfusion at a time before routine screening for hepatitis B virus (HBV).
- Hepatitis C virus (HCV) infection[4]
- Male sex
References
- ↑ Stone JH (October 2002). “Polyarteritis nodosa”. JAMA. 288 (13): 1632–9. PMID 12350194.
- ↑ Guillevin L, Mahr A, Callard P, Godmer P, Pagnoux C, Leray E; et al. (2005). “Hepatitis B virus-associated polyarteritis nodosa: clinical characteristics, outcome, and impact of treatment in 115 patients”. Medicine (Baltimore). 84 (5): 313–22. PMID 16148731.
- ↑ Hasler P, Kistler H, Gerber H (1995). “Vasculitides in hairy cell leukemia”. Semin Arthritis Rheum. 25 (2): 134–42. PMID 8578313.
- ↑ Ramos-Casals M, Muñoz S, Medina F, Jara LJ, Rosas J, Calvo-Alen J; et al. (2009). “Systemic autoimmune diseases in patients with hepatitis C virus infection: characterization of 1020 cases (The HISPAMEC Registry)”. J Rheumatol. 36 (7): 1442–8. doi:10.3899/jrheum.080874. PMID 19369460.
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