Vertigo primary prevention
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Zehra Malik, M.B.B.S[2]
Overview
There are no established measures for the primary prevention of vertigo, as it occurs as a symptom of underlying pathology.
Primary Prevention
- In some diseases controlling risk factors or triggering factors can prevent the disease hence preventing the symptoms:[1][2][3]
- Immunosuppression can provoke Herpes zoster oticus.
- Upper respiratory viral illness can lead to acute vestibular neuritis.
- Drug-induced vertigo: Dose reduction or discontinuation of the medication in patients presenting with vertigo may decrease the future incidence.
- Recognized triggers including altered sleep patterns, chocolate, red wine, ripened/aged cheese, can provoke vestibular migraine.
- Head injury can trigger epileptic vertigo.
- Increased stress can cause psychological vertigo.
- Changes in head position can provoke vertigo in acute labyrinthitis, benign positional paroxysmal vertigo, cerebellopontine angle tumor, multiple sclerosis, perilymphatic fistula.
- Perilymphatic fistula can be triggered by loud noises, changes in the ear pressure, excessive straining, head trauma.
References
- ↑ Hanley K, O’Dowd T, Considine N (2001). “A systematic review of vertigo in primary care”. Br J Gen Pract. 51 (469): 666–71. PMC 1314080. PMID 11510399.
- ↑ Derebery MJ (1999). “The diagnosis and treatment of dizziness”. Med Clin North Am. 83 (1): 163–77, x. doi:10.1016/s0025-7125(05)70095-x. PMID 9927968.
- ↑ Rosenberg ML, Gizzi M (2000). “Neuro-otologic history”. Otolaryngol Clin North Am. 33 (3): 471–82. doi:10.1016/s0030-6665(05)70221-8. PMID 10815031.
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