Health Dictionary
Reliable, easy-to-read patient information on common medical conditions — written in plain language, free of jargon, and grounded in current evidence. Browse the most frequent diagnoses, understand what they mean for everyday life, and learn which steps and questions help you have a focused conversation with your doctor.
Allergic colitis epidemiology and demographics
Introduction
Overview
Overview
The exact prevalence of allergic colitis is unknown. Prevalence of food protein-induced allergic proctocolitis (FPIAP) has been reported to range from a low of 16% to a high of 64% among infants with rectal bleeding. FPIAP is the most common cause of non-infectious colitis in infancy. Allergic colitis is mainly a disease of infants, with onset usually in the first two to three months of life. There is a slight male predominance (50–61.6%) for allergic colitis.
Epidemiology and Demographics
Epidemiology and Demographics
Prevalence and Incidence
The exact prevalence and incidence of allergic colitis is unknown.
- Prevalence of food protein-induced allergic proctocolitis (FPIAP) has been reported to range from a low of 16% to a high of 64% among infants with rectal bleeding[1][2][3]
- A population based study reported a prevalence of 1.6 in 1000 children under 1 year had cow’s-milk protein induced rectal bleeding[4]
- The incidence of milk FPIES was estimated as 0.34% of the newborn population in a population based study[4]
- 60% of infants with FPIAP are babies on exclusive breastfeeding[1][5]
- FPIAP is the most common cause of non-infectious colitis in infancy[6]
Age
Allergic colitis is mainly a disease of infants, with onset usually in the first 2-3 months of life. An adolescent form may develop in adolescence or early adulthood.[1][2][3][7]
Gender
There is a slight male predominance (50–61.6%) for allergic colitis.[8][9]
Race
There is no racial predilection for allergic colitis.
References
References
- ↑ 1.0 1.1 1.2 Nowak-WÄ™grzyn A (2015). “Food protein-induced enterocolitis syndrome and allergic proctocolitis”. Allergy Asthma Proc. 36 (3): 172–84. doi:10.2500/aap.2015.36.3811. PMC 4405595. PMID 25976434.
- ↑ 2.0 2.1 Arvola T, Ruuska T, Keränen J, Hyöty H, Salminen S, Isolauri E (2006). “Rectal bleeding in infancy: clinical, allergological, and microbiological examination”. Pediatrics. 117 (4): e760–8. doi:10.1542/peds.2005-1069. PMID 16585287.
- ↑ 3.0 3.1 Xanthakos SA, Schwimmer JB, Melin-Aldana H, Rothenberg ME, Witte DP, Cohen MB (2005). “Prevalence and outcome of allergic colitis in healthy infants with rectal bleeding: a prospective cohort study”. J Pediatr Gastroenterol Nutr. 41 (1): 16–22. PMID 15990624.
- ↑ 4.0 4.1 Elizur A, Cohen M, Goldberg MR, Rajuan N, Cohen A, Leshno M; et al. (2012). “Cow’s milk associated rectal bleeding: a population based prospective study”. Pediatr Allergy Immunol. 23 (8): 766–70. doi:10.1111/pai.12009. PMID 23050491.
- ↑ Lake AM (2000). “Food-induced eosinophilic proctocolitis”. J Pediatr Gastroenterol Nutr. 30 Suppl: S58–60. PMID 10634300.
- ↑ Sekerkova A, Fuchs M, Cecrdlova E, Svachova V, Kralova Lesna I, Striz I; et al. (2015). “High Prevalence of Neutrophil Cytoplasmic Autoantibodies in Infants with Food Protein-Induced Proctitis/Proctocolitis: Autoimmunity Involvement?”. J Immunol Res. 2015: 902863. doi:10.1155/2015/902863. PMC 4592904. PMID 26484355.
- ↑ Alfadda AA, Storr MA, Shaffer EA (2011). “Eosinophilic colitis: epidemiology, clinical features, and current management”. Therap Adv Gastroenterol. 4 (5): 301–9. doi:10.1177/1756283X10392443. PMC 3165205. PMID 21922029.
- ↑ Lozinsky AC, Morais MB (2014). “Eosinophilic colitis in infants”. J Pediatr (Rio J). 90 (1): 16–21. doi:10.1016/j.jped.2013.03.024. PMID 24131740.
- ↑ Mehr S, Frith K, Campbell DE (2014). “Epidemiology of food protein-induced enterocolitis syndrome”. Curr Opin Allergy Clin Immunol. 14 (3): 208–16. doi:10.1097/ACI.0000000000000056. PMC 4011623. PMID 24686277.
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Qasim Salau, M.B.B.S., FMCPaed [2]