Health Dictionary
Reliable, easy-to-read patient information on common medical conditions — written in plain language, free of jargon, and grounded in current evidence. Browse the most frequent diagnoses, understand what they mean for everyday life, and learn which steps and questions help you have a focused conversation with your doctor.
Epilepsy primary prevention
Introduction
overview
overview
Effective measures for the primary prevention of epilepsy include reducing the chance of possible epilepsy causes to happen including Head trauma, Hypoglycemia, Cerebral palsy, Electrolyte disturbances, fever, drug abuse and Vitamin deficiency and reduce the seizure triggers such as sleep deprivation, fever, light flashing, hyperventilation, alcohol, physical stress such as physical exercise or illness, psychological stress and depression.
Primary Prevention
Primary Prevention
- Effective measures for the primary prevention of epilepsy include:
- In healthy people, we can prevent epilepsy by reducing the chance of possible epilepsy causes to happen such as:
- In patients who already have epilepsy, we can reduce the seizure triggers such as:[8][9][10]
- Sleep deprivation
- Fever
- Light flashing
- Hyperventilation
- Alcohol
- Physical stress such as physical exercise or illness
- Psychological stress
- Depression
References
References
- ↑ Annegers JF, Coan SP (October 2000). “The risks of epilepsy after traumatic brain injury”. Seizure. 9 (7): 453–7. doi:10.1053/seiz.2000.0458. PMID 11034867.
- ↑ Dafoulas GE, Toulis KA, Mccorry D, Kumarendran B, Thomas GN, Willis BH, Gokhale K, Gkoutos G, Narendran P, Nirantharakumar K (February 2017). “Type 1 diabetes mellitus and risk of incident epilepsy: a population-based, open-cohort study”. Diabetologia. 60 (2): 258–261. doi:10.1007/s00125-016-4142-x. PMID 27796422.
- ↑ Diaconu G, Burlea M, Grigore I, Frasin M (2003). “[Epilepsy in different types of cerebral palsy]”. Rev Med Chir Soc Med Nat Iasi (in Romanian). 107 (1): 136–9. PMID 14755984.
- ↑ Riggs JE (February 2002). “Neurologic manifestations of electrolyte disturbances”. Neurol Clin. 20 (1): 227–39, vii. PMID 11754308.
- ↑ Shinnar S, Glauser TA (January 2002). “Febrile seizures”. J. Child Neurol. 17 Suppl 1: S44–52. doi:10.1177/08830738020170010601. PMID 11918463.
- ↑ Cendes F, Andermann F, Carpenter S, Zatorre RJ, Cashman NR (January 1995). “Temporal lobe epilepsy caused by domoic acid intoxication: evidence for glutamate receptor-mediated excitotoxicity in humans”. Ann. Neurol. 37 (1): 123–6. doi:10.1002/ana.410370125. PMID 7818246.
- ↑ Keyser A, De Bruijn SF (1991). “Epileptic manifestations and vitamin B1 deficiency”. Eur. Neurol. 31 (3): 121–5. doi:10.1159/000116660. PMID 2044623.
- ↑ Løyning Y (August 1993). “[Seizures and traffic risks]”. Tidsskr. Nor. Laegeforen. (in Norwegian). 113 (18): 2231. PMID 8362382.
- ↑ Thapar A, Kerr M, Harold G (January 2009). “Stress, anxiety, depression, and epilepsy: investigating the relationship between psychological factors and seizures”. Epilepsy Behav. 14 (1): 134–40. doi:10.1016/j.yebeh.2008.09.004. PMID 18824131.
- ↑ LEVIN M (February 1950). “The pathogenesis of narcoleptic and epileptic seizures occurring under acute emotional stress”. J. Nerv. Ment. Dis. 111 (2): 101–8. PMID 15409992.
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Fahimeh Shojaei, M.D.