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Reliable, easy-to-read patient information on common medical conditions — written in plain language, free of jargon, and grounded in current evidence. Browse the most frequent diagnoses, understand what they mean for everyday life, and learn which steps and questions help you have a focused conversation with your doctor.

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Esophagitis epidemiology and demographics

Introduction

Overview

Overview

In the USA and Europe, the prevalence of esophagitis ranges from low of 10,000 per 100,000 persons to high of 20,000 per 100,000 people. In Asia, the prevalence of esophagitis is 5,000 per 100,000 people. In the USA, the incidence of esophagitis is 5,400 per 100,000 persons. In Europe, the incidence of esophagitis is 840 per 100,000 persons.The prevalence of esophagitis increases with age. Men and women are affected equally by esophagitis. There is no racial predilection for esophagitis.

Epidemiology

Epidemiology

Prevalence

  • In the USA and Europe, the prevalence of esophagitis ranges from low of 10,000 per 100,000 persons to high of 20,000 per 100,000 people.[1][2][3][4][5][6][7]
  • In Asia, the prevalence of esophagitis is 5,000 per 100,000 people.
  • The prevalence of EoE is approximately 50-100 per 100,000 individuals worldwide.

Incidence

  • In the USA, the incidence of esophagitis is 5,400 per 100,000 persons.
  • In Europe, the incidence of esophagitis is 840 per 100,000 persons.
  • The incidence of EoE is approximately 10 per 100,000 individuals worldwide.
Demographics

Demographics

Age

  • The prevalence of esophagitis increases with age.
  • Esophagitis affects all age groups but it affects more the people older than 40 years.
  • Patients of all age groups may develop EoE.

Gender

  • Men and women are affected equally by esophagitis.
  • Males are more commonly affected by EoE than females.

Race

  • There is no racial predilection for esophagitis.
  • EoE usually affects individuals of the white race.
References

References

  1. ↑ Dellon ES (2014). “Epidemiology of eosinophilic esophagitis”. Gastroenterol. Clin. North Am. 43 (2): 201–18. doi:10.1016/j.gtc.2014.02.002. PMC 4019938. PMID 24813510.
  2. ↑ Soon IS, Butzner JD, Kaplan GG, deBruyn JC (2013). “Incidence and prevalence of eosinophilic esophagitis in children”. J. Pediatr. Gastroenterol. Nutr. 57 (1): 72–80. doi:10.1097/MPG.0b013e318291fee2. PMID 23539047.
  3. ↑ Sperry SL, Crockett SD, Miller CB, Shaheen NJ, Dellon ES (2011). “Esophageal foreign-body impactions: epidemiology, time trends, and the impact of the increasing prevalence of eosinophilic esophagitis”. Gastrointest. Endosc. 74 (5): 985–91. doi:10.1016/j.gie.2011.06.029. PMC 3951006. PMID 21889135.
  4. ↑ Cianferoni A, Spergel JM (2015). “Eosinophilic Esophagitis and Gastroenteritis”. Curr Allergy Asthma Rep. 15 (9): 58. doi:10.1007/s11882-015-0558-5. PMID 26233430.
  5. ↑ Furuta GT, Katzka DA (2015). “Eosinophilic Esophagitis”. N. Engl. J. Med. 373 (17): 1640–8. doi:10.1056/NEJMra1502863. PMC 4905697. PMID 26488694.
  6. ↑ Kocsis D, Tulassay Z, Juhász M (2015). “[Dietary and pharmacological aspects of eosinophilic esophagitis]”. Orv Hetil (in Hungarian). 156 (23): 927–32. doi:10.1556/650.2015.30164. PMID 26027600.
  7. ↑ El-Serag HB, Sweet S, Winchester CC, Dent J (2014). “Update on the epidemiology of gastro-oesophageal reflux disease: a systematic review”. Gut. 63 (6): 871–80. doi:10.1136/gutjnl-2012-304269. PMC 4046948. PMID 23853213.

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Ajay Gade MD[2]]