IgA nephropathy physical examination

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Dildar Hussain, MBBS [2]

Overview

Patients with IgA nephropathy usually appear normal and usually have no significant clinical finding upon physical examination. However, some of the patients may present with low-grade fever, high blood pressure with normal pulse pressure, and pitting edema of the lower extremities in the late stage if the patient develops ESRD.

Physical Examination

Physical examination of patients with IgA nephropathy is usually normal.^[1]^[2]^[3]^[4]^[5]

Appearance of the Patient

Patients with IgA nephropathy usually appear normal.

Vital Signs

Low-grade fever
High blood pressure with normal pulse pressure

Skin

Skin examination of patients with IgA nephropathy is usually normal.

HEENT

HEENT examination of patients with IgA nephropathy is usually normal.

Neck

Neck examination of patients with IgA nephropathy is usually normal.

Lungs

Pulmonary examination of patients with IgA nephropathy is usually normal.

Heart

Cardiovascular examination of patients with IgA nephropathy is usually normal.

Abdomen

Abdominal examination of patients with IgA nephropathy is usually normal.

Back

Back examination of patients with IgA nephropathy is usually normal.

Genitourinary

Genitourinary examination of patients with IgA nephropathy is usually normal.

Neuromuscular

Neuromuscular examination of patients with IgA nephropathy is usually normal.

Extremities

Pitting edema of the lower extremities develops in late stage, if the patient develops ESRD

References

↑ Galla JH (February 1995). “IgA nephropathy”. Kidney Int. 47 (2): 377–87. PMID 7723227.
↑ Donadio JV, Grande JP (September 2002). “IgA nephropathy”. N. Engl. J. Med. 347 (10): 738–48. doi:10.1056/NEJMra020109. PMID 12213946.
↑ Hall CL, Bradley R, Kerr A, Attoti R, Peat D (October 2004). “Clinical value of renal biopsy in patients with asymptomatic microscopic hematuria with and without low-grade proteinuria”. Clin. Nephrol. 62 (4): 267–72. PMID 15524056.
↑ Topham PS, Harper SJ, Furness PN, Harris KP, Walls J, Feehally J (June 1994). “Glomerular disease as a cause of isolated microscopic haematuria”. Q. J. Med. 87 (6): 329–35. PMID 8041865.
↑ Gutierrez, E.; Gonzalez, E.; Hernandez, E.; Morales, E.; Martinez, M. A.; Usera, G.; Praga, M. (2006). “Factors That Determine an Incomplete Recovery of Renal Function in Macrohematuria-Induced Acute Renal Failure of IgA Nephropathy”. Clinical Journal of the American Society of Nephrology. 2 (1): 51–57. doi:10.2215/CJN.02670706. ISSN 1555-9041.

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[pmid7723227-1] Galla JH (February 1995). “IgA nephropathy”. Kidney Int. 47 (2): 377–87. PMID 7723227.

[pmid12213946-2] Donadio JV, Grande JP (September 2002). “IgA nephropathy”. N. Engl. J. Med. 347 (10): 738–48. doi:10.1056/NEJMra020109. PMID 12213946.

[pmid15524056-3] Hall CL, Bradley R, Kerr A, Attoti R, Peat D (October 2004). “Clinical value of renal biopsy in patients with asymptomatic microscopic hematuria with and without low-grade proteinuria”. Clin. Nephrol. 62 (4): 267–72. PMID 15524056.

[pmid8041865-4] Topham PS, Harper SJ, Furness PN, Harris KP, Walls J, Feehally J (June 1994). “Glomerular disease as a cause of isolated microscopic haematuria”. Q. J. Med. 87 (6): 329–35. PMID 8041865.

[GutierrezGonzalez2006-5] Gutierrez, E.; Gonzalez, E.; Hernandez, E.; Morales, E.; Martinez, M. A.; Usera, G.; Praga, M. (2006). “Factors That Determine an Incomplete Recovery of Renal Function in Macrohematuria-Induced Acute Renal Failure of IgA Nephropathy”. Clinical Journal of the American Society of Nephrology. 2 (1): 51–57. doi:10.2215/CJN.02670706. ISSN 1555-9041.

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