Mycobacterium avium complex infection
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
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Historical Perspective
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Classification
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Pathophysiology
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
Pathophysiology
Etiologic Agent:
Mycobacterium avium complex (comprising M. avium and M.intracellulare).
Transmission:
Although the mode of transmission is unclear, MAC is most likely environmentally acquired.
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Causes
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Differentiating Mycobacterium avium complex infection from other Diseases
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Epidemiology and Demographics
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
Epidemiology and Demographics
Not reportable. Population-based data available for Houston and Atlanta metropolitan areas suggest a rate of 1/100,000/year. Incidence is decreasing among HIV- infected patients as a result of new treatment modalities e.g., combination therapy with nucleoside reverse transcriptase inhibitors and protease inhibitors, as well as antimycobacterial prophylaxis. Incidence is decreasing because of changes in treatment for HIV-infected patients; however, antimicrobial resistance may be increasing.
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Risk Factors
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Risk Factors
HIV-infected persons. Rarely in children or nonimmuno-compromised persons.
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Natural History, Complications and Prognosis
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Diagnosis
Diagnosis
History and Symptoms | Physical Examination | Laboratory Findings | Chest X Ray | CT | MRI | Echocardiography or Ultrasound | Other Imaging Findings | Other Diagnostic Studies
Treatment
Treatment
Medical Therapy | Primary Prevention | Secondary Prevention | Cost-Effectiveness of Therapy | Future or Investigational Therapies
Acknowledgements
Acknowledgements
The content on this page was first contributed by: C. Michael Gibson, M.S., M.D. Template:SIB
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