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Myocardial rupture pathophysiology

Introduction

Overview

Overview

The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. Other causes of rupture include cardiac trauma, endocarditis (infection of the heart),[1][2] cardiac tumors, infiltrative diseases of the heart,[1] and aortic dissection.

Pathophysiology

Pathophysiology

Images shown below are courtesy of Professor Peter Anderson DVM PhD and published with permission © PEIR, University of Alabama at Birmingham, Department of Pathology

Myocardial Rupture of the Free Wall

Ventricular Septal Rupture

References

References

  1. ↑ 1.0 1.1 Lin TH, Su HM, Voon WC, Lai HM, Yen HW, Lai WT, Sheu SH. (2006). “Association between hypertension and primary mitral chordae tendinae rupture”. Am J Hypertens. 19 (1): 75–9. PMID 16461195.
  2. ↑ de Diego C, Marcos-Alberca P, Pai RK. (2006). “Giant periprosthetic vegetation associated with pseudoaneurysmal-like rupture” (PDF). Eur Heart J. 27 (8): 912. PMID 16569654.


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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]