
Health Dictionary
Reliable, easy-to-read patient information on common medical conditions — written in plain language, free of jargon, and grounded in current evidence. Browse the most frequent diagnoses, understand what they mean for everyday life, and learn which steps and questions help you have a focused conversation with your doctor.
Reactive arthritis epidemiology and demographics
Introduction
Overview
Overview
The incidence of reactive arthritis following a gastrointestinal or genitourinary infection is approximately 3-27 cases per 100,000 individuals worldwide. The prevalence of reactive arthritis is approximately 30-40 cases per 100,000 individuals worldwide. Reactive arthritis commonly affects young adults in the age group of 18-35 years of age. Men are more commonly affected with reactive arthritis than females with male to female ratio of approximately 4:1. There is no racial predilection to reactive arthritis.
Epidemiology and Demographics
Epidemiology and Demographics
Incidence
- The incidence of reactive arthritis following a gastrointestinal or genitourinary infection is approximately 3-27 cases per 100,000 individuals worldwide.[1]
- The incidence rate of reactive arthritis varies with the underlying infection.[2]
- The incidence rate of reactive arthritis following Campylobacter infection is 900 per 100,000 cases.
- The incidence rate of reactive arthritis following Salmonella and Shigella infection is 1200 per 100,000 cases.
Prevalence
- The prevalence of reactive arthritis is approximately 30-40 cases per 100,000 individuals worldwide.[3][4][5]
Age
- Patients of all age groups may develop reactive arthritis.
- Reactive arthritis commonly affects young adults in the age group of 18-35 years of age.
Race
- There is no racial predilection to reactive arthritis.
Gender
- Reactive arthritis affects both men and women. However, men are more commonly affected than females.
- The male to female ratio is approximately 4:1.
References
References
- ↑ Townes JM, Deodhar AA, Laine ES, Smith K, Krug HE, Barkhuizen A, Thompson ME, Cieslak PR, Sobel J (December 2008). “Reactive arthritis following culture-confirmed infections with bacterial enteric pathogens in Minnesota and Oregon: a population-based study”. Ann. Rheum. Dis. 67 (12): 1689–96. doi:10.1136/ard.2007.083451. PMID 18272671.
- ↑ Ajene AN, Fischer Walker CL, Black RE (September 2013). “Enteric pathogens and reactive arthritis: a systematic review of Campylobacter, salmonella and Shigella-associated reactive arthritis”. J Health Popul Nutr. 31 (3): 299–307. PMC 3805878. PMID 24288942.
- ↑ Townes JM (January 2010). “Reactive arthritis after enteric infections in the United States: the problem of definition”. Clin. Infect. Dis. 50 (2): 247–54. doi:10.1086/649540. PMID 20025528.
- ↑ Rohekar S, Pope J (July 2009). “Epidemiologic approaches to infection and immunity: the case of reactive arthritis”. Curr Opin Rheumatol. 21 (4): 386–90. doi:10.1097/BOR.0b013e32832aac66. PMID 19373091.
- ↑ Hannu T (June 2011). “Reactive arthritis”. Best Pract Res Clin Rheumatol. 25 (3): 347–57. doi:10.1016/j.berh.2011.01.018. PMID 22100285.
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Akshun Kalia M.B.B.S.[2]
