Health Dictionary
Reliable, easy-to-read patient information on common medical conditions — written in plain language, free of jargon, and grounded in current evidence. Browse the most frequent diagnoses, understand what they mean for everyday life, and learn which steps and questions help you have a focused conversation with your doctor.
Wild-type (senile) amyloidosis physical examination
Introduction
Overview
Overview
Physical examination of patients with wild-type (senile) amyloidosis can be significant for the condition in question and can also translate the variety of pathologies as a part of aging and age-related comorbidities.
Physical Examination
Physical Examination
Physical examination of patients with wild-type (senile) amyloidosis can be significant for the condition in question and can also translate the variety of pathologies as a part of aging and age-related comorbidities.
Appearance of the Patient
- Patients with wild-type (senile) amyloidosis appear senile.
- Exact appearance of the patients depend upon the severity of the condition.
Vital Signs
Skin
HEENT
- HEENT examination of patients with wild-type (senile) amyloidosis is usually normal.
Neck
Lungs
- Pulmonary examination of patients with wild-type (senile) amyloidosis is usually normal.
Heart
- Third heart sound
- Fourth heart sound
- Arrhythmias [2]
- Auscultation may also reveal rales, heart murmurs, and/or other pathologic heart sounds.
- In patients with valvular heart disease involvement, murmurs of mitral and tricuspid regurgitation may be heard.
Abdomen
- Hepatomegaly may be present in the setting of right heart failure
- Ascites may be present in the setting of right heart failure
- Splenomegaly is rare
Back
- Back examination of patients with wild-type (senile) amyloidosis is usually normal.
Genitourinary
- Genitourinary examination of patients with wild-type (senile) amyloidosis is usually normal.
Neuromuscular
Extremities
References
References
- ↑ Belinda Ng, Lawreen H. Connors, Ravin Davidoff, Martha Skinner & Rodney H. Falk (2005). “Senile systemic amyloidosis presenting with heart failure: a comparison with light chain-associated amyloidosis”. Archives of internal medicine. 165 (12): 1425–1429. doi:10.1001/archinte.165.12.1425. PMID 15983293. Unknown parameter
|month=ignored (help) - ↑ Rapezzi C, Perugini E, Salvi F, Grigioni F, Riva L, Cooke RM, Ferlini A, Rimessi P, Bacchi-Reggiani L, Ciliberti P, Pastorelli F, Leone O, Bartolomei I, Pinna AD, Arpesella G, Branzi A (2006). “Phenotypic and genotypic heterogeneity in transthyretin-related cardiac amyloidosis: towards tailoring of therapeutic strategies?”. Amyloid : the International Journal of Experimental and Clinical Investigation : the Official Journal of the International Society of Amyloidosis. 13 (3): 143–53. doi:10.1080/13506120600877136. PMID 17062380. Retrieved 2012-02-13. Unknown parameter
|month=ignored (help) - ↑ Belinda Ng, Lawreen H. Connors, Ravin Davidoff, Martha Skinner & Rodney H. Falk (2005). “Senile systemic amyloidosis presenting with heart failure: a comparison with light chain-associated amyloidosis”. Archives of internal medicine. 165 (12): 1425–1429. doi:10.1001/archinte.165.12.1425. PMID 15983293. Unknown parameter
|month=ignored (help)
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Sabawoon Mirwais, M.B.B.S, M.D.[2]Jesus Rosario Hernandez, M.D. [3]Raviteja Guddeti, M.B.B.S. [4]; Aarti Narayan, M.B.B.S [5]; Cafer Zorkun, M.D., Ph.D. [6]; Lakshmi Gopalakrishnan, M.B.B.S. [7]